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ATP-Sensitive Potassium Channel-Deficient Mice Show Hyperphagia but Are Resistant to Obesity |
Yeul Bum Park, Yun Jung Choi, So Young Park, Jong Yeon Kim, Seong Ho Kim, Dae Kyu Song, Kyu Chang Won, Yong Woon Kim |
Diabetes Metab J. 2011;35(3):219-225. Published online June 30, 2011 DOI: https://doi.org/10.4093/dmj.2011.35.3.219 |
ATP-Sensitive Potassium Channel-Deficient Mice Show Hyperphagia but Are Resistant to Obesity Methods for Characterizing Disease-Associated ATP-Sensitive Potassium Channel Mutations Production and purification of ATP-sensitive potassium channel particles for cryo-electron microscopy The Potential Role of Activating the ATP-Sensitive Potassium Channel in the Treatment of Hyperphagic Obesity Sulfonylurea Receptor Transmembrane Domain Zero Mutations that Disrupt Full Length and Minimal ATP-Sensitive Potassium Channel Properties Disruption of Kir6.2-containing ATP-sensitive potassium channels impairs maintenance of hypoxic gasping in mice Sulfonylureas Correct Trafficking Defects of Disease-causing ATP-sensitive Potassium Channels by Binding to the Channel Complex Channel activators regulate ATP-sensitive potassium channel (KIR6.1) expression in chick cardiomyocytes Reduction in ATP-sensitive potassium channel-mediated antinociception in diabetic mice ATP-sensitive potassium channel (KATP)-mediated antinociception is reduced in diabetic mice. |