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Role of Activation of NF- B and AP-1 by Oxidative Stress in Atherosclerosis in Diabetic Patients. |
Chul Sik Kim, Geun Taek Lee, Jina Park, Min Ho Cho, Joo Young Nam, Jong Suk Park, Dol Mi Kim, Chul Woo Ahn, Bong Soo Cha, Sung Kil Lim, Kyung Rae Kim, Hyun Chul Lee |
Diabetes Metab J. 2004;28(4):255-264. Published online August 1, 2004 |
Th-P15:163 Increased macrophage oxidative stress via-NADPH-oxidase activation and impaired cholesterol fluxes in diabetic mice: Role of cellular glucose uptake Increased oxidative stress in diabetic patients as a risk factor for atherosclerosis 3.P.44 Atherosclerosis and oxidative stress in diabetic patients The role of oxidative stress and NF-κB activation in late diabetic complications Oxidative stress in families of type 1 diabetic patients Role of endogenous secretory RAGE (esRAGE) in defending against plaque formation induced by oxidative stress in type 2 diabetic patients Redox Control of Gene Expression by Eukaryotic Transcription Factors NF-κB, AP-1 and SRF/TCF Effect of alpha-lipoic acid supplementation on oxidative stress markers and antioxidative defense in patients with diabetic neuropathy Epigenetic Basis of Oxidative Stress in Diabetic Coronary Atherosclerosis: A Shift in Focus from Genetic Prerogative Glutathione depletion exacerbates impairment by oxidative stress of phosphoinositide hydrolysis, AP-1, and NF-κB activation by cholinergic stimulation |